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Whoever is striving to grow muscle mass without taking the risk of undesired aftereffects caused by the somatropin hormoneprolactin.”
There’s a lot to digest (or read into).
I’ve taken many, many pills in the past few years.
I’ve tried multiple creams, gels, etc. to make my body more like this one.
And I’ve gotten ripped.
Most studies seem to support the “good hormone” theory — but how do we translate this to a human being’s perspective?
Here’s a look at what the good hormone theory actually says:
Prolactin (prolactin is the hormone that produces a surge of prolactin when a woman starts to ovulate) reduces your growth hormone production (growth hormone is needed for muscle mass).
Growth hormone has a number of different functions. It’s involved in muscle contractility, body composition, and fat redistribution.
I think this sounds like a nice rationale, and you should follow this model when looking at human growth hormone studies. In addition, you should look at the results of “prolactin reduction of growth hormone secretion” (a study I’ve recently read) and be willing to consider two hypotheses for the cause.
Hypothesis: Low prolactin results in lean mass loss.
The first hypothesis (based on the “good hormone” hypothesis) is the one that’s most frequently discussed. Prolactin-deficient folks have the opposite of lean mass — as discussed here.
The second hypothesis (based on this one) is “higher than normal levels of prolactin, and thus high body fat.”
The second hypothesis has not been observed in the research community.
However, a study by Fitts et al. (2007) found that an increase in prolactin was associated with a significant increase in body fat.
So, is this a good model for understanding what we want to see in a human being?
The science: Yes. I think the concept that hormone levels are influenced by food intake is the best way for anyone to deal with the problem of human growth hormone being lower than normal.
It does seem like the right idea. We would all benefit from some kind of a growth hormone treatment for people with growth hormone deficiencies.
But how do we look at this model in a way that doesn’t reinforce the “good hormone” myth that is perpetuated about our bodies?
This is where the good hormone hypothesis gets complicated.
There are several
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